Nephrotic syndrome will not cause dyslipidemia change?

Nephrotic syndrome in patients with hyperlipidemia mechanism are still not clear. Nephrotic syndrome patients often result from the large number of urine protein plasma protein decreased, hypoproteinemia may be compensatory to make proteins, especially if very low density lipoprotein lipoprotein (VLDL) accelerated synthesis, which can be transformed into low-density lipoprotein (LDL), thereby increasing LDL, this is probably the reasons for the formation of hyperlipidemia.
Therefore, the majority of patients with nephrotic syndrome increased plasma lipid, or even a fasting plasma can be emulsion. Although a variety of blood lipids can be increased, but the largest increase in the neutral lipid, cholesterol and triglycerides are increased, the extent of decline and plasma albumin was negatively correlated. Early blood cholesterol more than 7.15 cents mole / liter, serious increased to 25.86 cents Moore / or about cholesterol and phospholipid ratio increased to an average of 1.28 (0.95 for the average normal value). Yellow skin can occur in patients with tumor.
Nephrotic syndrome hyperlipidemia lasted too long, there may be atherosclerosis, thrombosis and embolism. Researchers have observed that chronic renal insufficiency patients of normal triglyceride, lipoprotein lipase activity decreased triglyceride decomposition obstacles At the same time, patients with chronic renal insufficiency blood triglyceride levels increased vascular lesions increased the incidence of, and I seriously treating hyperlipidemia. However, these patients with hyperlipidemia on the response of poor diet therapy should be used in combination with the bile acid resins and nicotinic acid lipid-lowering treatment.