Parietal lobe syndrome

Outlined in the frontal parietal, occipital before. After back into central and supramarginal gyrus, angular gyrus and the top lobule. Parietal lobe syndrome main obstacles to feel the symptoms, but there are still obstacles like missing structural disorders, movement disorders and other symptoms. Cause pathogenesis one. Injury : Traumatic brain injury is especially Top fractures, which often caused acute damage to the parietal lobe, consciousness obstacle. According to statistics, the top depressed skull fracture depressed skull fracture 66%. 2. Tumor : parietal lobe tumor can be used not structural. Vestibular stimulation of tumor may appear nystagmus. 3. Vascular Disease : middle cerebral artery disease when symptoms emerge parietal group agraphia, Reading and Gerstmannn loss syndrome. A clinical performance. Sensory dysfunction : parietal lobe lesions, there will be limitations of sensory epilepsy and a sophisticated sensory dysfunction. Performance of acupuncture, electric shock, the feeling of pain and abnormal attack entity McGREGOR, Tinel positioning Cox and skin loss. 2. Dyskinesia : parietal lobe lesions, either alone or hemiplegia paralysis, can be seen deep hyperreflexia, muscle tension was not significantly higher. 3. Muscular Dystrophy : occurs in the contralateral lesions proximal upper extremity, accompanied shoulder dislocation, was Aran - Duchenne muscular atrophy. 4. Ataxia : parietal lobe lesions, deeply feeling obstacles ataxia, eyes when ataxia Obviously, eyes closed aggravating. Found in the contralateral limb or limbs part. 5. Visual and eye movement disorders : There visual deformation, bigger or smaller, distant or recent changes. Also, there will be the visual phenomenon stranded and lasting sexual gaze backward. 6. Body Image : Body image disturbance to the parietal lobe lesions special symptoms. Performance of the patients to understand the structure of autologous obstacles. Clinical hemiplegia missing Note disorders, hemiplegia denied syndrome, phantom limb phenomenon, the body about disorientation and limbs occurred illusion misinterpreted, and so on. 7. Structure apraxia : patients with the right objects, architecture, design, graphics, and other spatial relations and the lack of three-dimensional concept, not for permutations and combinations. 8.Gerstmann syndrome : angular gyrus, supramarginal gyrus and the parietal-occipital transitional parts of the lesion, appeared calculated not, fail to recognize fingers, not understanding about side and unable to write, "with four missing." Differential diagnosis (1) cerebrovascular disease (cerebrovascular disease) cerebrovascular disease hemiplegia caused by the disease, physically, there may be muscle atrophy, mainly upper limb muscles of the hands, such as size of fish muscle, bone interosseous muscle atrophy. Forearm muscles may also suffer and lead to "ape hands" or "claw-shaped hand." Muscular atrophy confined to the shoulder girdle, especially in the deltoid muscle, often shoulder dislocation. But this muscular atrophy is clear hemiplegic cerebral vascular disease after a few weeks or a few months there, and physically shrinking Kang tendon reflexes, Hypertonia. (2) syringomyelia (symptom) syringomyelia be out side or both sides of upper extremities The muscular atrophy, is a progressive muscular atrophy, or reduce tendon reflexes disappear from the horn of the spinal cord damage caused. Segmental ipsilateral skin pain, temperature Cox diminish or disappear, and deep feeling, sensitivity and retained that feeling of separation barrier. The location of the skin and bones sometimes there Nutrition disorders. Moreover, as syringomyelia occur in the cervical and upper thoracic pulp, tissue associated with Horner's sign. (3) the parietal lobe tumors (parietal lobe tumor) parietal lobe tumors induced muscle atrophy, confined to the more proximal upper extremity, often shoulder dislocation, is Aran - Duchenne muscular atrophy. In addition to muscle atrophy accompanied cortical sensory dysfunction, upper extremity and hand autonomic barriers, hand-purple skin temperature change, abnormal sweating and subcutaneous tissue or bone abnormalities.