Glossopharyngeal, Vagus, vice hypoglossal nerve damage and

Overview glossopharyngeal, vagal, vice hypoglossal nerve and were starting medulla oblongata, medulla from the act also close neighbors, after the group called the cranial nerves, often simultaneously involved. Which glossopharyngeal and vagus nerve is a common nuclear threshold and close around the pathway, the clinical s merger damage, have palsy. But primary glossopharyngeal neuralgia is a separate involvement. Etiopathologic disease triggered Glossopharyngeal, Vagus, hypoglossal nerve and cause a lot of damage, it is common to the basilar invagination. Central pillow integration deformities, cerebellopontine angle tumor, Polyneuritis, cranial neuritis, meningitis, trauma, etc.. Because of its different etiology pathogenesis different, such as inflammatory infiltration and demyelination changes, and so on. A clinical performance. Glossopharyngeal and vagus nerve paralysis : palsy can be caused, for the performance of dysarthria and dysphagia. Dysarthria for hoarseness, there nasometry, slurring in speech, or even completely Aphonia. Dysphagia can dysphagia, and are choking, severe totally swallowing. Can detect one or both could not mention the soft palate, uvula side were visible to the contralateral Mobile. Pharyngeal feeling and gag reflex retardation or loss. 2. Nerve paralysis : by the sternocleidomastoid muscle paralysis, low muscle tone, muscular atrophy, turn their difficulties. Side nerve paralysis performance for the first not to the contralateral side, trapezius muscle paralysis, low muscle tone, muscular atrophy, and can not shrug high. 3. Hypoglossal nerve paralysis : Shengua ipsilateral to the side of distortion, muscle atrophy, muscle tremor and other fibers. 4. Glossopharyngeal neuralgia : See Section 2 of Chapter IV. Differential diagnosis (1) acute inflammatory demyelinating polyneuropathy (acute inflammutory axonal polynearopathy) first symptom, often unable limbs symmetry with gloves Motao was feeling kind obstacles. How cranial nerve roots while impaired, or a single cranial nerve damage to the main. Regular involvement of the glossopharyngeal, and vagus, the Deputy and facial nerve, for the first performance backwards, hoarseness, choking, facial paralysis. CSF is a protein-cell separation. (2) - wan occipital malformation (congenital anomalies of atlanto - oc cpital region) are more or eat torticollis, but the occasion low, facial asymmetry. Slow onset, symptoms of nervous system checks to the main group after the cranial nerves, cerebellum, spinal cord, neck and neck nerve compression symptoms. The most common headache, dizziness, neck pain pillow, often due to the head or manual activities caused by ataxia, unsteady gait, Nystagmus relative styles, individual cases are unclear articulation, hoarseness, dysphagia, difficulty in breathing, apnea, muscle atrophy. sternocleidomastoid muscle weakness, facial paralysis, deafness and other. Skull films for the skull base pressure measurement can trace showed abnormality. (3) cerebellopontine angle tumors (cerebellopontine angle tumors) slow onset patients, the performance of the cerebellopontine angle syndrome and intracranial hypertension clinical symptoms, when the tumor development, Article IX of oppression, Ⅹ, Ⅺ cranial nerve, which can cause difficulty swallowing, choking cough, hoarseness, ipsilateral gag reflex diminish or disappear. soft palate palsy, trapezius and sternocleidomastoid muscle weakness. Hypoglossal nerve were rare. CT and MRI can find tumor growth location. (4) intracranial metastases (intracranial metastatic tumors) Since the skull base nasopharyngeal carcinoma or sarcoma can be abused where cranial nerve paralysis. Short course, it will be bloody nasal secretions, cervical lymph node metastasis more. ENT examination and biopsy will be confirmed. (5) arachnoid adhesions (arachnoid membrane adhesim) before the disease has made more Thermal history, but also a history of chronic meningitis, long course and there will be the cranial nerve palsy symptoms, such as dysphagia, hoarseness, dysarthria unclear, facial paralysis, CSF white blood cell count increased inspection. (6) Central (pseudo) bulbar palsy (central bulbar paralysis) again said on motor neurons or nuclear palsy, bilateral cortical - medullary beam damage caused. Visibility in the cerebral arteriosclerosis, multiple infarction, such as encephalitis. The main clinical manifestations of speech slurring in speech, swallowing difficulties lighter, the tongue is not due to food shipped to the throat caused. Gag reflex still exist, and the mandibular reflex, such as palm chin brainstem reflex reflector can hyperthyroidism; Cone beam can be accompanied by the levy, and a strong cry-laugh.