Wednesday, March 12, 2008

Cerebellar syndrome

Cerebellum outlined in the posterior fossa medullary above, the dorsal brain stem, cerebral hemispheres of the ventral occipital, and cerebellar screen was covered. Cerebellum into cerebellar vermis and the cerebellar hemispheres of the two. According species occur on the order of cerebellar cerebellum into ancient, old and new cerebellum cerebellum. Cerebellar syndrome prominent characteristic of muscle hypotonia, ataxia and tremor. Cause pathogenesis one. Trauma : cerebellar traumatic brain injury more rare, simply more traumatic for the cerebellum cerebellum penetrating injury. Cerebellar symptoms caused by traumatic coma obscure. 2. Cerebrovascular disease : cerebellar dentate nucleus artery rupture cerebellar hemorrhage caused half-length behind the cerebellum devastating symptoms and signs. Ipsilateral lesion on the lower limb ataxia, reduce muscle tension, thus reflecting diminished or disappeared. 3. Tumor : cerebellar hemisphere tumor may arise intracranial hypertension symptoms and signs and bilateral abducens nerve palsy. 4. Degeneration disease : cerebellar hemispheres and vermis cortical atrophy, degeneration of nerve cells and decrease presenting highlights of the performance of standing and walking instability and mild language barriers. 5. Infections : acute infection caused small encephalitis, acute cerebellar ataxia. 6. Poisoning : acute alcoholism and long-term use of large doses of phenytoin may arise cerebellar ataxia. A clinical performance. Ataxia : patients with basilar widened when standing up, body shaking instability, often on the side of dumping difficult to stand or not. When intoxicated men while walking gait. 2. Identified from the bad : cerebellar lesions in patients with right distance, velocity and movement are underestimating the strength needed to assess abnormal, there pointed nose test, a change of trial closed. 3. Reduce muscle tension : cerebellar hemisphere lesion ipsilateral lesion appeared to reduce muscle tension, physically abnormal posture. there pendulum-like knee jerk. 4. Intention tremor : motor frequent coarse irregular tremor, static disappears. 5. Speech and writing obstacles : Four patients with ambiguous language, a poet-like language or explosive speech. When her writing is not sharp, serrated is, the more characters to write the greater spacing ranges. 6. Nystagmus : performance for horizontal, vertical and rotary nystagmus, a level nystagmus seen more often. 7. Postural tremor : for patients to sit up and running posture when there postural tremor, and runs through the entire process of action, static disappeared. Differential diagnosis (1) of cerebellar hemorrhage (cerebellar hemorrhage) sudden onset, the first symptoms of headache, vomiting, and other acute symptoms of increased intracranial pressure and dizziness. Most patients at the onset of conscious, there gait ataxia, a small number of patients with sudden paralysis. Pupil normal or smaller, and both can be asymmetric. Two common gaze more contralateral to the lesion, a small number of people with nystagmus. Cerebellar hemorrhage in the diagnosis based on : sudden onset, vomiting, low muscle tone, in contrast to a small hole or asymmetric corneal reflex slow or disappear. (2) cerebellar tumor (cerebellar tumor) was particularly prevalent in children, in the cerebellar hemisphere tumors disappearing main symptoms of ataxia campaign, early lesions appeared ipsilateral limb ataxia. Intention tremor, reduce muscle tension, the one after the vestibular labyrinth symptoms and headache, vomiting, palilledema levy for the three intracranial hypertension symptoms, and was accompanied abducens nerve palsy, slow pulse, the baby before the Hang prominent, Separation skull and scalp vein dilatation. Tumor suppression structure near the facial numbness, corneal reflex reduce or loss. Late weak physical damage and the pyramidal tract signs. (3) cerebellar abscess (cerebellar abscess) is a common intracranial suppurative disease. The main symptoms are systemic symptoms : fever, chills, headache, vomiting, neck resistance flu, Kornig's levy positives. Intracranial hypertension in the general two-three weeks there. Mainly located in the occipital headache, neck or to the prefrontal radiotherapy. Significant lesion side of ataxia, hypotonia low nystagmus. Late after cranial nerve palsy. When the affected brain stem, vertigo, headache, vomiting, ataxia, pronunciation and Dysphagia, horizontal nystagmus with Part IV, Ⅶ right cranial nerve palsy. (4) olive-ponto-cerebellar atrophy (olivary body-pons - cerebellu m atrophy OPCA) symptoms of the disease for the first double-leg balance disorders, was intoxicated men while gait. Limbs, trunk and cerebellar hemisphere ataxia. Cerebellar symptoms gradually worsened at the same time, the Department of extrapyramidal symptoms, increased muscle tone, the lack of facial expression, sometimes similar kind of paralysis agitans tremor occurred also myoclonic and operation of the pyramidal tract symptoms and signs. Sometimes dysarthria, dysphagia, limbs mild muscle atrophy, muscle fibrillation and nystagmus. (5) Fridereich's ataxia syndrome (friedreich's tataxia) for the post-merger cerebellar lesions REQUEST A lesion. Obviously ataxia, static coordination disorder more obvious than the campaign, trunk limb ataxia more prominent. Generally start with the lower extremity developed symptoms appeared when standing and walking instability around deflection. Reduce muscle tension, bad measure, nystagmus, Romberg patients. Deep sensory dysfunction. Often Babinski sign, tendon reflexes disappear, cerebellar dysarthria, with adequate varus deformity and scoliosis. (6) Small acute encephalitis (acute cerebellitis) ~ found in a 4-year-old child, Duality can be found in adults aged around 30. Acute ataxia and dysarthria. Often disturbance of consciousness, sometimes mildly pyramidal tract signs of damage. (7) multiple sclerosis (multiple sclerosis) of the disease was acute or subacute onset. Early, there may be dizziness and pain. Cranial nerve obstacles to the performance of oculomotor nerve palsy with Pulley also emerging nystagmus, to the level of, Rotating or vertical. Chang cerebellar lesions caused by unclear pronunciation, voice, ranging from the pros. Deep sensory dysfunction obviously serious when there were fluent sensory disorders, usually feel symptoms within a few weeks to ease. Cone beam can damage into spastic paralysis. and appeared euphoric color-based psychiatric symptoms. With painful cramps and internuclear ophthalmoplegia performance characteristics.

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