Balint's syndrome

Also known as outlined cortex gaze palsy. Cause pathogenesis of bilateral parietooccipital lesions, especially in the bilateral posterior cerebral artery and its branches in spasm or obstruction. Clinical manifestations eyeball free movement disappear, eye movement and visual attention disorders, but to preserve spontaneity and reflection eye movement, guide speech difficulties, agraphia, ideas, sports apraxia symptoms. As the watch center cortex interrelated, so cortex side of the eye damage caused by paralysis of time is very short, About a few hours to three days to resume, bilateral cortical damage induced paralysis more attention to the long-lasting nature. Occipital lesions rarely watch hub preoptic caused paralysis. Differential diagnosis (1) cerebral infarction (cerebral 362002) of the middle cerebral artery embolization have Balint's syndrome. Other suppliers on the hypothalamus and brain stem of blood, when there infarct area with the contralateral to the Blind, Main side of the hemisphere except when infarct cortex sensory dysfunction, but also appeared aphasia, alexia, agraphia, agnosia, such as language and parietal lobe syndrome. Features for its high incidence of age, a sudden onset than resting incidence generally occurred without hemiplegia. Infarct area as small, timely and appropriate treatment, the symptoms are expected to resume, otherwise leave from sequelae. (2) brain tumor (brain tumor) happened parietooccipital area in the tumor with gliomas and meningiomas styles. Early and often headache history of epilepsy treatment, with the growth of tumors, headaches and nausea increased, vomiting and palilledema increased intracranial pressure and other symptoms. Balint levy is a late, CT enhanced lesions visible, obvious signs of mass.