Duodenal ulcer and gastric ulcer treatment principle What are the different

Duodenal ulcer and gastric ulcer on the same digestive ulcers. Its pathogenesis, there are a lot in common, so there are a lot of common ground. In the treatment of gastrointestinal ulcer drugs broadly divided into two categories: those for the protection of mucous membrane. Application of such drugs more Japanese imports of Mai Zilin - S; German imports will rule the stomach, stomach Tarivid; China-made music in the stomach, stomach-speed music, Corning stomach, the stomach and Thailand on March 9, Di music, such as Sidel . Mucosa in the protection of the application, duodenal ulcer and gastric ulcer is no different from, the medication method, roughly the same dose. Taken on the different treatment is another major point - drugs that attack on the factors inhibiting drug. The representatives of such drugs: cimetidine, ranitidine, famotidine and omeprazole, and so on, and they are by inhibiting gastric acid to achieve therapeutic purposes. We know that because the cancer may have gastric ulcer, in particular the low-acid ulcer cancer more likely, in patients with gastric ulcer attack factor inhibiting drugs use of short course of caution, mostly through medication before and after endoscopy From living tissues to verify whether there are cancerous. However, duodenal ulcer in the use of such drugs, their treatment can be extended. This is in the treatment of the two main principles on the different points. As for the prevention of gastric ulcer medication, most do not have to attack factor inhibitors, which advocated using mucosal protection; duodenal ulcer is not this limit, they can be used

What is cured after gastric ulcer recurrence

Gastric ulcer recurrence rate is high, up around 80 percent. Because of gastric ulcer is a chronic, recurrent natural ease and the tendency, so most people think, alleviate the symptoms of acute ulcer easy, difficult to prevent ulcer recurrence. It can be said that the prevention of the recurrence of ulcers, treatment of ulcer disease is the most difficult part, because it not only requires us to understand the incidence of gastric ulcer factors. And requires that we understand their recurrence, but at present these issues have not yet fully understand, so to resolve recurrent problems are certain difficulties. The recurrence of a common factor: ulcer cure does not adhere to medication maintenance therapy, physical fatigue, anxiety, stress, insomnia, excessive smoking, improper diet, drugs and irritation of the infection. If perfect, too acid, spicy foods to stimulate long-term use of aspirin, Bao Tai Song, hormone drugs. The following measures to reduce relapse frequency and extent of martial law. (1) the full treatment of active ulcer disease. (2) avoid alcohol, tobacco, coffee, Nongcha, stimulating drugs and food. (3) adequate rest and sleep. (4) to avoid long-term mental stress, anxiety and so on. (5) positive treatment of respiratory tract infections and other infections between. (6) the slightest signs of recurrence, or a mild symptoms, immediately strict treatment

Why would children suffering from canker

Ulcer disease in children with duodenal ulcer most common. No major law and diet, eating in moderation does not, Puyinpusi, and other factors. Bianchi Bian some children playing, some parents or children in the table on the teaser or a meal reprimanded the children. Some children get up at night, in order to catch up with classes, eating breakfast also Gubu Shang. Some children or to rush to Tanwan homework Mission Lun Tun date to be rice Yanxia, often also finished with three two-soaked rice or steamed bread, open blisters. If things continue this way to easily lead to ulcer disease. Because of outdated completely wrong meal so that stomach emptying a high degree of hunger, causing stomach reverse peristalsis, the expansion of blood vessels caused by gastric mucosa edema, acid damage to the local inflammatory ulcer. Food Guobao, a high degree of expansion of the antrum, gastrin secretion reduce the ability of the pyloric sphincter, caused by the reflux of duodenal, easy to damage the barrier function gastric ulcer caused. Children in the canker of reasons, are no more than can be summarized as follows: (1) bad eating habits, uneven hungry enough, not of life. (2) breakfast or Langtunhuyan too lax, or non-Jin breakfast. (3) a cold drink and snack food. (4) Guoduopilao, mental tension. (5) had gastritis not adhere to treatment, from chronic gastritis or gastric ulcer disease into the development of inflammation.

Gout stones can occur in the ureter and bladder?

Gout occurs in kidney stones there are two main reasons: (1) the excretion of uric acid in the blood mainly through the kidneys, about 75 to 80 percent of uric acid from the kidneys, about 20 percent of uric acid into the intestine through the biliary Road, with the droppings from. Other organs such as lung, pancreas, and so almost non-ranked urinary acid, uric acid in kidney-the deposition of a kidney stone. Particularly high concentration of uric acid in the blood over the discharge capacity of the kidney, kidney stones more prone to gout. (2) uric acid deposition and local environmental organizations the pH (the PH value). When the local Piansuan, that is, when the low value of HP uric acid deposition to form stones when the local side at the base, that is at high PH value, not in some uric acid deposition. Normal blood PH value of 7.35 to 7.45 (neutral). Liver, lung, heart, brain and other organizations of similar PH value and blood, and uric acid secretion is not the place, so uric acid deposition difficult to form stones. Formation of urine and kidney is the place where the urine PH value in general about 5.5 to 6, acid, and uric acid in urine from the main, so easy in the deposition and formation of kidney stones. Sometimes in the ureter and bladder stones forming the deposition. In a few cases, gout-can also be in the vessel wall, Endocardial and bile duct to form, but such cases are rarely seen.

How early detection of gout

Early detection of gout the most simple and effective method is to detect blood uric acid concentrations. The crowd on a large scale survey of serum uric acid can be found in time hyperuricemia, which is early detection and early prevention is of great significance to have gout. In the present conditions there is no large-scale blood uric acid test of the circumstances, at least the following staff to deal with the routine blood uric acid test: (1) over 60 years old, both male and female and whether obesity. (2) obesity in middle-aged men and women after menopause. (3) hypertension, atherosclerosis, coronary heart disease, cerebrovascular disease (such as cerebral infarction and cerebral hemorrhage) patients. (4) Diabetes (mainly type II diabetes). (5) the reasons for not prescribed arthritis, especially over the middle-aged patients with arthritis, as a single characteristic. (6) kidney stones, kidney stones and in particular the multiple bilateral kidney stone patients. (7) members of the family history of gout. (8) long-term meat, and drinking habits of middle-aged people over. All the above are listed in the case of any one of the people, shall take the initiative to go to the hospital to do the laboratory examination of the gout, and early discovery hyperuricemia and gout, do not wait until there has been a typical clinical symptoms (such as subcutaneous Gout stones) to seek treatment before. If the initial inspection normal serum uric acid, gout and can not be easily ruled out the possibility of hyperuricemia. After should regularly review, the annual health check at least once. This will enable the early detection rate of gout increased substantially.

What are the types of gout

Generally speaking, adult men who have acute arthritis, especially the thumb of those affected, must be highly vigilant against the possibility of suffering from gout, if their blood has higher concentrations of uric acid, can be diagnosed as gout. Typical symptoms of individual patients and the concentration of uric acid in the blood is not high, they repeated blood test, examination or take joint fluid, sodium crystallization of uric acid found, can also be diagnosed as gout. Gout has two primary and secondary. As early as the 5th century BC, the Greek physician Hippocrates, gout is a genetic disease. Modern medicine also believe that the primary gout and the enzyme and metabolic defects, and other relevant, most are family history, foreign coverage is 10% -25%, there are similar reports of domestic. The disease was first developed symptoms of age from more than 10 years, until about 80 years old, the average age of 40-year-old from top to bottom. 95% of patients are male adults. Many patients before the onset of symptoms, the concentration of uric acid in the blood have increased significantly. Female patients accounted for about 5 percent, most of the patients in the period after menopause symptoms. Most secondary gout is due to chronic hemolytic anemia, leukemia and myeloma, malignant neoplasms, hyperparathyroidism, such as various kidney a result of renal dysfunction caused by. Gout generally appear first time, often in the original have been a few years after the onset. In addition, high-calorie, high-purine diet and drinking can also be induced gout. According to clinical manifestations of gout can be divided into three categories: ① acute gouty arthritis; ② tophi and chronic arthritis; ③ gouty nephropathy.

Gout can cause kidney damage?

Gout can appear kidney damage. According to statistics, gout patients are 20 to 25 per cent of uric acid kidney, and the autopsy confirmed that a kidney disease almost 100%. It includes gouty nephropathy, acute obstruction of the urinary tract and kidney stones. (1) gouty nephropathy persistent hyperuricemia, 20% in clinical performance on the nephropathy, after several years or more may have a glomerular and tubular damage to the development of a small number of urine Duzheng. Urate the incidence of kidney after gouty joint damage, and is closely related with the disease and treatment. Research shows that uric acid salt nephropathy and gouty arthritis unrelated to the extent that the mild arthritis patients may have renal disease, arthritis and severe kidney patients is not necessarily abnormal. Early mild unilateral or bilateral pain, swelling and later a mild to moderate hypertension. Urinary acid, intermittent or sustained proteinuria, generally no more than + +. Almost all renal function decline in enrichment, a nocturnal enuresis, and more urine and urinary relatively low density. About five to ten years after the kidney increase, for further development of uremia, about 17% -25% died of renal failure. (2) urolithiasis gout patients urinary acid, thus increasing concentration of uric acid in urine, with smaller stones from the urine, but often without feeling, urinary sediments shows that small brown sand; larger stones can be obstructive Ureter caused hematuria and renal colic, because of a urinary infection secondary to poor pyelonephritis. Huge stones can cause renal pelvis beacon deformation, hydronephrosis. X uric acid stones simply not the online image, when uric acid, sodium and calcium stones that X online shadow. (3) Acute obstructive nephropathy found in blood uric acid in urine and uric acid increased significantly, it is because a large number uric acid crystal tubular obstruction caused by extensive. Gout often and hypertension, hyperlipidemia, atherosclerosis, coronary heart disease and type 2 diabetes. Gout in older persons in the cause of death, cardiovascular factors far more than renal insufficiency. However, gout and cardiovascular diseases there is no direct causal link between, but with both obesity and dietary factors are related.

Gout condition of the development process in several period

Gout is a lifelong disease, it's condition development of the entire process can be divided into the following four: (1) hyperuricemia period: also known as pre-gout, in this period the patient can no clinical symptoms of gout, only for the performance of serum uric acid increased. (2) early gout: This period from hyperuricemia developed from. Prominent symptoms of acute gouty arthritis attack. In acute arthritis disappeared after the attack joints can be fully returned to normal, not left over from the damage, but can be repeated attack. This usually subcutaneous the formation of tophi, there is no obvious kidney disease such as uric acid nephropathy and the formation of kidney stones, kidney function was normal. (3) gout medium-term: this period because of repeated gouty arthritis caused by acute injury to a joint varying degrees of bone destruction and dysfunction, chronic gout. Gout can be a subcutaneous stone, may have uric acid nephropathy and the formation of kidney stones, kidney function may be normal or slightly decreased. (4) advanced gout: a clear joint deformity and dysfunction, the increase in the number of subcutaneous tophi, the size of the increase can Pokui a white crystalline salt urine. Uric acid nephropathy and kidney stones to develop and kidney function decreased significantly, there azotemia and uremia.

Four dangerous complications of gout

Patients suffering from gout, in sugar and fat metabolism will be significantly decreased, making it easy with all kinds of serious diseases: [Diabetes] Diabetes and gout because both are in metabolic disorders caused by disease, it is easy with the patients. Diabetes is the regulation of blood sugar because of a lack of insulin hormone, resulting in a continuous state of high blood sugar, and uric acid and blood glucose levels between the very relevant, usually high uric acid, blood glucose levels will be higher. [Hypertension / high blood lipids: Most patients with gout is more body fat, excessive accumulation of body fat tends to cause arteriosclerosis and hypertension, and patients with gout because of bias on the daily intake of dietary fat, high-calorie food, so in the neutral fat content is very high , Cholesterol values are generally have more than the normal standard, hyperlipidemia is a good one of the ethnic groups. [Myocardial infarction / heart disease - a narrow Gout patients prone to cardiovascular arteriosclerosis of the situation, resulting in the blood can not be fully served on the heart, blood circulation dysfunctional, cause heart disease or myocardial infarction narrow the probability of particularly high, especially on the original with hyperlipidemia Gout patients are more prone to heart disease. [Cerebral vascular obstacles: The same is guided by the problem in atherosclerosis, the difference in the brain occur. Its symptoms include headache, dizziness, numbness or tingling hands and feet, serious, the patients have lost their sense of danger and even death, the patient volume, in addition to angiography, the brain needs to be done CT, MRI examination.

Tophi arthritis and chronic gout

Suffering from chronic gout tophi and more chronic arthritis patients, the crystallization of uric acid in the body will be deposited in the cartilage and synovial membrane, and soft tissue, the formation of tophi, and blood in the higher concentration of uric acid, with The longer period of disease, may be deposited, the more tophi, and sometimes will affect the blood vessels and kidneys, causing serious renal failure, nephrotic more serious and difficult to create a vicious cycle of uric acid excretion, the Tophi Also the more deposition. Often the site of deposition tophi many, including the ears, hands, elbows, Achilles tendon, ankle or toe, and sometimes even give rise to local ulcer and difficult healing, and even the latest addition to requiring surgery. Patients and cause serious deformation or chronic joint symptoms of severe deformation of the foot when the patient may cause serious problems on the shoes. In addition, the risk of kidney stones occur with increased serum uric acid concentrations increase, and often lead to renal disease, renal failure may be subject to after hemodialysis, which is caused gout patients died one of the main causes of death.

Secondary hyperuricemia, or gout

Secondary hyperuricemia, or gout that is a minor clinical manifestations, secondary to any genetic or acquired pathological process. Clinical mainly divided into two categories: A. generate excessive uric acid 1) excessive amount of high-purine food intake, 2) bone marrow proliferative diseases: multiple myeloma, 3) lymphoproliferative disease: infectious mononucleosis Cell Histiocytosis, 4) hemolytic anemia, 5) tumors, 6) psoriasis, 7) the cumulative lying liver disease: found I, III, V, VI, 8) fructose intake, 9) is not hereditary fructose Tolerance, 10) hypoxemia and organizations hypoperfusion, 11) intense muscle movement, 12) acute and chronic alcohol poisoning, B. reduce uric acid excretion 1) renal insufficiency, 2) ketoacidosis, 3) lactic acid Acidosis, 4) tubular absorb increased uric acid salts, 5) dehydrating agent and diuretic, 6) hyperparathyroidism, 7) parathyroid mechanism may be, 8) hypothyroidism, 9) hypertension, 10) Drugs: ethambutol, pyrazinamide, nicotinic acid, acetyl acid water Yang, 11) lead poisoning. 12) patients with cancer chemotherapy and radiotherapy, 13) adrenal mechanism may be, 14) renal diabetes insipidus, 15) hunger.

Gouty arthritis and gout What is the relationship between

Gout is a metabolic disorder and beautiful reduce the excretion of uric acid caused by a group of diseases. The clinical performance are: asymptomatic hyperuricemia, gout arthritis stone, urinary stone and renal disease. Gouty arthritis is when uric acid salts in the articular cartilage or synovial deposition, leading to synovial tissue and the inflammatory response. Hyperuricemia is the most important biochemical basis of gout, but is not synonymous with gout. As improvement of living standards, changes in the structure of diet, and many affected uric acid excretion of drugs (diuretics) the extensive use of hyperuricemia is in fact the incidence of the adult male is 5% -7%, the researchers statistics About 5% -12% of hyperuricemia in patients with gout for the eventual development. When the blood uric acid level of more than 420 micro-Mount / l, arthritis and gout in uric acid kidney stones, or the risk of a significant increase

Hyperuricemia and gout What's the difference between

Hyperuricemia is uric acid in the blood more than a normal state. Many reasons can cause blood uric acid salt content increased, and gout is one of the most common causes. Gout is in the long-term hyperuricemia circumstances, result in human organs and tissues in diseases. The main result of gouty arthritis, gout and kidney disease, gout and kidney stones, gout and heart disease, hypertension and gout, and other serious complications. Hyperuricemia can also say is a prelude to gout, but not necessarily able to evolve into gout disease, the patients have gout and hyperuricemia.

Tophi can take stone surgery

According to statistics, gout patients in about 20% to 30% of body surface of a stone, the stone occurred in the vicinity of the joints and limbs, and joint function in patients with serious decline, maimed, or even loss of self-care ability. Some patients stone not long after surgery wound healing, some doctors do not want to do this surgery. Gout can then stone surgery stones » How surgery » Tophi formation of tophi is because long-term patients increased serum uric acid, uric acid formation of sodium (MSU), deposited in various tissues and organs formed stones. In addition to internal organs outside the brain can be a stone to see most of the kidney stone. Surface stones mainly occurred in the vicinity of the joints and limbs, the stone sooner or later, the growth rate and increased blood levels of uric acid, hyperuricemia duration, and acute arthritis in the same number of joint attack was positively correlated. Stone serious bone and joint, hand or foot bones of several pieces of stone were all occupied. If the initial stones in the soft tissue, the skin can thinning, Pokui, MSU crystallization of the outflow or small stones to form ulcers and sinus, repeated bacterial infections, break a few years or 10 years can not be healed, the merger also osteomyelitis Are not uncommon. So as soon as possible surgery stones. ① preoperative preparation before the surgery should be the first of serum uric acid to normal or near normal levels, after a result of uric acid avoid excessive impact on healing. ② need to physically do the surgery X-ray imaging, X-ray easily through the stone, but the soft tissue in the stone often can be seen surrounding the density slightly higher than the whole of the massive image. X-ray through the little bone, stone density was significantly lower than bone density, X-ray film common wormhole-like, Chuanzao kind, the kind of low-density cellular bone defects, serious one or several pieces of bone and parts All missing. Gout is a metabolic syndrome ③ a disease, should be actively treating hypertension, diabetes and renal dysfunction and other merger-related diseases, to be in stable condition after surgery. Surgical operation is primarily aimed at joints and near the stone: ① where stone diameter of more than 2 cm or more than a stone so that the pool in uric acid increased significantly, the impact of descending uric acid treatment. ② have ulcers, sinus or osteomyelitis formed to break long-term, non-union. ③ stones affect joint function, significantly restricted the activities of patients. If surgery is not invaded stone and bone joints, regardless of whether the merger ulcer and sinus, the majority can and will be coated with stone removed, the incision in five to ten days to heal. Such as stone and bone and joint deep, large piece of stone removed, should no longer use keys, and other equipment will be scraping the remnants of stone fragments as far as possible eliminate, or stone debris will continue in the overflow from the wound, healing effects, drainage should be done at this time Treatment, more than in January after the drainage is not sealed, should be done in the second removal. A small number of serious bone damage, the need to buy bones, joints integration, and the truncated limb, so to promote tophi surgery sooner the better. ① down after dealing with uric acid, a small stone after stone in the blood return to the residue dissolved so that the blood uric acid increased rapidly, if not promptly and effectively to drop acid treatment, after the majority of people in the non-operative joint acute arthritis Some multi-joint alternating repeated incidence. At this point in addition to eat food rich in purine, down uric acid to increase the amount of drugs, and for seven days after the previous day to a daily service 1 ~ 3 mg colchicine, can significantly reduce acute arthritis. ② strengthen hypertension, coronary heart disease, diabetes, kidney disease and other diseases related to the treatment of these diseases under control.

Tophi can take stone surgery

According to statistics, gout patients in about 20% to 30% of body surface of a stone, the stone occurred in the vicinity of the joints and limbs, and joint function in patients with serious decline, maimed, or even loss of self-care ability. Some patients stone not long after surgery wound healing, some doctors do not want to do this surgery. Gout can then stone surgery stones » How surgery » Tophi formation of tophi is because long-term patients increased serum uric acid, uric acid formation of sodium (MSU), deposited in various tissues and organs formed stones. In addition to internal organs outside the brain can be a stone to see most of the kidney stone. Surface stones mainly occurred in the vicinity of the joints and limbs, the stone sooner or later, the growth rate and increased blood levels of uric acid, hyperuricemia duration, and acute arthritis in the same number of joint attack was positively correlated. Stone serious bone and joint, hand or foot bones of several pieces of stone were all occupied. If the initial stones in the soft tissue, the skin can thinning, Pokui, MSU crystallization of the outflow or small stones to form ulcers and sinus, repeated bacterial infections, break a few years or 10 years can not be healed, the merger also osteomyelitis Are not uncommon. So as soon as possible surgery stones. ① preoperative preparation before the surgery should be the first of serum uric acid to normal or near normal levels, after a result of uric acid avoid excessive impact on healing. ② need to physically do the surgery X-ray imaging, X-ray easily through the stone, but the soft tissue in the stone often can be seen surrounding the density slightly higher than the whole of the massive image. X-ray through the little bone, stone density was significantly lower than bone density, X-ray film common wormhole-like, Chuanzao kind, the kind of low-density cellular bone defects, serious one or several pieces of bone and parts All missing. Gout is a metabolic syndrome ③ a disease, should be actively treating hypertension, diabetes and renal dysfunction and other merger-related diseases, to be in stable condition after surgery. Surgical operation is primarily aimed at joints and near the stone: ① where stone diameter of more than 2 cm or more than a stone so that the pool in uric acid increased significantly, the impact of descending uric acid treatment. ② have ulcers, sinus or osteomyelitis formed to break long-term, non-union. ③ stones affect joint function, significantly restricted the activities of patients. If surgery is not invaded stone and bone joints, regardless of whether the merger ulcer and sinus, the majority can and will be coated with stone removed, the incision in five to ten days to heal. Such as stone and bone and joint deep, large piece of stone removed, should no longer use keys, and other equipment will be scraping the remnants of stone fragments as far as possible eliminate, or stone debris will continue in the overflow from the wound, healing effects, drainage should be done at this time Treatment, more than in January after the drainage is not sealed, should be done in the second removal. A small number of serious bone damage, the need to buy bones, joints integration, and the truncated limb, so to promote tophi surgery sooner the better. ① down after dealing with uric acid, a small stone after stone in the blood return to the residue dissolved so that the blood uric acid increased rapidly, if not promptly and effectively to drop acid treatment, after the majority of people in the non-operative joint acute arthritis Some multi-joint alternating repeated incidence. At this point in addition to eat food rich in purine, down uric acid to increase the amount of drugs, and for seven days after the previous day to a daily service 1 ~ 3 mg colchicine, can significantly reduce acute arthritis. ② strengthen hypertension, coronary heart disease, diabetes, kidney disease and other diseases related to the treatment of these diseases under control.